Answer:
Nonsense mutation: it causes a premature stop codon, so the protein cannot be fully synthesized.
Missense mutation: it causes change on the aminoacid encoded, so it can cause a change in the protein structure if the new aminoacid doesn't have the same chemichal properties as the original.
Synonymous (silent) mutation: it causes no change, the same aminoacid is encoded.
Single nucleotide insertion or deletion: changes the entire structure of the protein because it shifts the reading frame.
Three nucleotide deletion: one aminoacid will no longer be part of the protein, if this aminoacid was located, for example, on the active site of an enzyme, the protein could lose its function.
Chromosomal translocation: it can break a gene in two, causing the protein to no longer be able to be synthesized, or it can change the transcription regulation because it is now under the effect of other regulating sites that result in a different transcription pattern.
Explanation:
What is the greatest danger to a patient who has had damage to the skin? excessive muscle contractions in the damaged area loss of oils produced by the skin infections in uncovered tissues damaged tissues entering the blood stream?
Answer:
If all the bacteria are removed from the earth, all the organisms are negatively affected.
Explanation: There are also some beneficial bacteria which helps in the nitrogen fixation called Nitrogen fixing bacteria such as Cyanobacteria and Azotobactor. These bacteria fixes atmospheric nitrogen into ammonium and nitrates form because plants are unable to use the atmospheric nitrogen, it only use nitrogen in ammonia and nitrates form. Plants absorb nitrates and form amino acid.
Answer:
umm you should translate to English your question
Answer:
Blood-cell cancer that begins in the bone marrow.
Explanation:
Chronic myeloid leukemia is caused by a rearrangement (translocation) of genetic material between chromosome 9 and 22. This translocation, written as t(9;22), fuses part of the ABL1 gene from chromosome 9 with part of the BCR gene from chromosome 22, creating an abnormal fusion gene called BCR-ABL1.
