Answer:Product side
Explanation:
The number of molecules is increasing
<h2>CNS </h2>
Explanation:
An example of a myelin producing cell in the CNS is oligodendrocyte
- The major function of oligodendrocytes is the formation of myelin
- Myelin acts as an insulator of axonal segments and is a prerequisite for the high velocity of nerve conduction
- Larger axons form thicker myelin
- During development, oligodendrocytes arise from precursors located in the sub-ventricular zone such as the sub-ventricular zone of the lateral ventricles for the cerebrum or the fourth ventricle for the cerebellum
- In the spinal cord, oligodendrocytes originate from the ventral regions of the neural tube and in the optic nerve they migrate into the nerve from the third ventricle
- It is the oligodendrocyte precursor cells which migrate to their destination where they then differentiate into the more mature oligodendrocytes
- The proliferation of the oligodendrocyte progenitor cells is controlled by a number of growth factors released predominantly from neurons but also from astrocytes such as platelet derived growth factor (PDGF) or fibroblast growth factor (FGF)
The ATP molecule is a high energy molecule. When a molecule of ATP breakdown, it produces chemical as well as thermal energy. The type of energy produced is mainly depends on the application for example, if the ATP breaks down to ADP, it produces thermal energy and if couples with a cellular process it produces chemical potential.
Our body needs energy to carry out cellular functions and the energy produced in a form of ATP is taken by cells to perform functions.
Answer:
The old idea that coronary heart disease is an infectious disease has gained popularity in recent years, and both viral and bacterial pathogens have been proposed to be associated with the inflammatory changes seen in atherosclerosis. Herpes group viruses, notably cytomegalovirus and herpes simplex type 1, have been associated with atherosclerosis and restenosis. Helicobacter pylori and dental infections have also been linked to atherogenesis, but the evidence seems to favor a respiratory, obligatory intracellular bacterium, Chlamydia pneumoniae. The association was originally found in seroepidemiological studies, but the actual presence of the pathogen in atherosclerotic lesions has been repeatedly demonstrated, and during past year the first successful animal experiments and encouraging preliminary intervention studies were published. The causal relationship has not yet been proven, but ongoing large intervention trials and continuing research on pathogenetic mechanisms may lead to the use of antimicrobial agents in the treatment of coronary heart disease in the future.
Explanation:
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