Answer:
allopatric speciation by dispersal
Explanation:
Allopatric speciation is a form of speciation (creation of new species) that occurs as a result of geographic isolation. This means that a part of population becomes physically separated from the initial main population. There is no gene flow between these two populations and as a result the two populations reach a high level of genetic divergence. They can no longer interbreed which means they become two different species (speciation).
One of the factors that contribute to the allopatric speciation is dispersal.
New populations evolve as result of mutation, genetic drift and natural selection.
Answer:
A.Glycogenesis: Glycogen synthase
B. Glucogenesis: Fructose 1,6 biphosphate phosphatase
C. Urea cycle : Carbamoyl phosphate synthetase
D.Fatty acid synthesis: Acetyl CoA carboxylase
E.Glycolysis : Phosphofructokinase 1
F. Pentose phosphate pathway: Glucose-6-phosphate dehydrogenase
Explanation:
A. Glycogen synthase converts glucose into glycogen during glycogenesis.
B. Fructose 1,6 biphosphate phosphatase catalyzes condensation of dihydroxyacetone phosphate (DHAP) and glyceraldehyde-3-phosphate during glucogenesis.
C. Carbamoyl phosphate synthetase I catalyses production of arbamoyl phosphate during urea cycle.
D. Carboxylase controls fatty acid metabolism.
E. The phosphofructokinase 1 is an important enzyme that regulate formation of two-phosphate sugar molecules during glycolysis.
F. Glucose-6-phosphate dehydrogenase participates in the pentose phosphate pathway. This pathway gives reducing energy to cells.
Answer:
the answer is D. All cells contain a nucleus
Answer:
Cyanide is considered a specific neurotoxin and is often associated with high levels of cellular calcium and inhibition of antioxidants, leading to the formation of reactive oxygen species (ROS) and consequent lipid peroxidation. But cyanide toxicity is mainly due to the iron (Fe (III) or Fe3 +) iron binding of the mitochondrial cytochrome oxidase enzyme of the electron transport chain. This enzyme mediates the transfer of electrons to molecular oxygen
. By blocking this enzyme, the cell is unable to use oxygen and uses an anaerobic metabolism reducing pyruvate to lactic acid (lactic fermentation), which results in tissue anoxia and the rapid development of lactic acidosis. There is also a change in cell metabolism as there is a depletion in energy production. So it causes cell death. This would be the mechanism to kill cancer cells. Although, cyanide cant be use to treat cancer because it kills all the cells (leading to death) and not only the cancer cells.
Explanation:
Some researchers founded that cyanide poison may have the role of being a powerful cancer fighter. It believes that they can relate the enzyme to cancer by looking for antibodies that can recognize a protein found only in certain cancers. The method would produce an agent that could potentially kill cancer cells and leave undamaged tissue healthy.
As for the deadly nature of cyanide, scientists say the amount produced by this technique iwould be sufficient only to kill the cancer target cell by a combined enzyme and antibody molecule. And a natural enzyme produced in the human liver could detoxify any cyanide that could escape the tumor. Although, cyanide is a drug that, even if ingest a small amount, could lead to death. Some studies are going on, but until now, theres no safety of taking cyanide to treat cancer cells.