It generally complained of flu-like symptoms (headache, fever, muscle pain, sore throat) followed closely by stomach pain, vomiting, and diarrhea, which sometimes progressed to the bruising and hemorrhaging that he was seeing in his current patient.
<span>It was the culmination of research in the 1930s and early 1940s at the Rockefeller Institute for Medical Research to purify and characterize the "transforming principle" responsible for the transformation phenomenon first described in Griffith's experiment of 1928: killed Streptococcus pneumoniae of the virulent strain type III-S, when injected along with living but non-virulent type II-R pneumococci, resulted in a deadly infection of type III-S pneumococci.</span>
The attached picture shows how bacteria gain antibiotic resistance. Firstly, a few individuals attain a
beneficial mutation in their genetic material that accords them the capability to survive in an antibiotic. The
individuals are hence able to survive and
reproduce more than those individuals without the mutation. There is, therefore, a genetic
shift in the population in favor of the resistant genotype. After generations, the
whole population becomes antibiotic resistant.
Answer: Roles of DNA polymerases and other replication enzymes. ... which require a template and a primer (starter) and synthesize DNA in the 5' to 3' direction. ... Topoisomerase also plays an important maintenance role during DNA replication.
Explanation:
hard to explain
I don't think so, because for every action there is an equal and opposite reaction.
Hope this helps! :)
