Answer:through carbon cycle
Explanation: look up for the word and also look at the diagrams for better understanding
Answer:
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Explanation:
The non-coding portions of DNA that are separated from the portions of DNA actually used during transcription are called. -exons. -introns.
Answer:
a
Explanation:
When a gene mutation occurs, the nucleotides are in the wrong order which means the coded instructions are wrong and faulty proteins are made or control switches are changed. The body can't function as it should. Mutations can be inherited from one or both parents. They are present in the egg and/ or sperm cells.
Answer:
Genes that are relatively close to one another on a chromosome will move with one another during the process of fertilization and meiosis. Thus, there will be a lined inheritance for these genes. On the other hand, genes that are found on different chromosomes will have independent inheritances because every pair of the homologous chromosomes will be positioned differently when the
y are arranged at the middle of the cell during the first meiotic division.
Explanation:
Genes that are relatively close to one another on a chromosome will move with one another during the process of fertilization and meiosis. Thus, there will be a lined inheritance for these genes. On the other hand, genes that are found on different chromosomes will have independent inheritances because every pair of the homologous chromosomes will be positioned differently when the
y are arranged at the middle of the cell during the first meiotic division.
Yes, it is true that Mammalian target of rapamycin pathway mutations cause hemimegalencephaly and focal cortical dysplasia.
Focal malformations of cortical development, including focal cortical dysplasia (FCD) and hemimegalencephaly (HME), are important causes of intractable childhood epilepsy.
Using targeted and exome sequencing on DNA from resected brain samples and non-brain samples from 53 patients with FCD or HME, we identified pathogenic germline and mosaic mutations in multiple PI3K/AKT pathway genes in 9 patients, and a likely pathogenic variant in 1 additional patient.
Our data confirm the association of DEPDC5 with sporadic FCD but also implicate this gene for the first time in HME. Our findings suggest that modulation of the mammalian target of rapamycin pathway may hold promise for malformation-associated epilepsy.
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