Answer:
Hello your question is incomplete attached below is the complete question
answer :
None of the progenator cells differentiate into type 2 cells. ( A )
Explanation:
Due to the loss-of- function mutation in the gene for the Notch receptor in the progenitor cell of C elegans , This causes The progenator cells for type 1 to be differentiated to type 1 cells but the cells for type 2 will not be differentiated.
hence the answer is : None of the progenator cells differentiate into type 2 cells.
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Answer:
In heterozygotic individuals, the gene for sickle cell anemia is protective against malaria.
Explanation:
Through the same mechanism that the mutation associated with sickle cell anemia produces the disease (altered hemoglobin structure), having a single copy of the mutation (that is, being heterozygotic) provides protection against malaria. This is believed to be due to the altered structure causing difficulty for the malaria parasite when it attempts to enter red blood cells as part of its life cycle.
Today, there are medications to successfully treat malaria, but it still killed 627,000 people in 2020. This represents a significant evolutionary pressure for genes that would prevent serious malarial illnesses. As malaria was and still is common in African regions, the result was a higher prevalence of sickle cell anemia in those of African descent.
Answer: Option B.
Neural crest and peripheral nervous system.
Explanation:
Neural crest are bilateral paired cells of the neural tube that arise from the ectoderm layer of the embryo. Thesescells move to different part of the body and differentiate into various cell types like melanocytes,cartilage and bone, smooth muscle, craniofacial, neurons, gangalia e.t.c. The neural crest running through neural tube develop into peripheral nervous system after birth. Peripheral nervous system consist of neurons and gangalia outside the nervous system The peripheral nervous system connect the central nervous system to organs, skin and limbs.