Answer:
B. Two groups of monerans-Eubacteria and Archaebacteria--differed significantly from one another
Explanation:
It was Carl Woese who divided the prokaryotes into 2 kingdoms, Eubacteria and Archaebacteria based on RNA studies. So. this led to additional 6th classification added to Whittaker's classification of 5 kingdoms.
Answer:
The G-T mismatches generally originate because of the spontaneous deamination of 5-methylcytosine to thymine. Therefore, correcting G-T to G≡C most probably preserves the original sequence.
In the DNA, the 5-methylcytosine usually goes through the process of deamination to thymine. This transformation may cause a G-T base pair or mismatch of nucleotide pairs. Generally, the mutation like C-T transition is common. The G-T base pair is identified by base excision repair proteins, which withdraws this mismatched base pair.
The repair system encourages methylation of cytosine as the mechanism of the regulation of transcription, which permits the turning off or on the process of transcription on the basis of the requirement of the cells. This also assists in preventing the mutation of DNA.
Answer:
Deleterious alleles appear sporadically in a population
Explanation:
A population with a deleterious allele will have no or few individuals that have the ability to pass along these traits. These alleles appear less in a population because of selective pressure but they are not always absent. The alleles appear less often but are are not always passed on and the others that are genetically fit are able to pass along their genes. The reason the population equilibrium is not zero is because these alleles do appear but they are not necessarily passed along. These individuals may not be able to reproduce or reach the age of reproduction.
Deleterious alleles appear more often, making individuals less fit genetically, i.e. they pass fewer copies of their genes to future generations. Put another way, natural selection purges the deleterious alleles.
When faced with dangers close to both sides of your path of travel you should create more space to the side with the most serious consequences. There are several dangers and obstacles on the roadway and Tips to Avoid them which include; a deer in the headlights,unidentified flying objects on the roads, slow moving vehicles, speed bumps, pedestrian and pet such as dogs, cats may be common obstacles. It is always important to weigh the extent of dangers while faced with dangers on both sides of the paths.
Answer:
Each mutant would be mated to wild type and to every other mutant to create diploid strains. The diploids would be assayed for growth at permissive and restrictive temperature. Diploids formed by mating a mutant to a wild type that can grow at restrictive temperatures identify the mutation as recessive. Only recessive mutations can be studied using complementation analysis. Diploids formed by mating two recessive mutants identify mutations in the same gene if the diploid cannot grow at restrictive temperature (non-complementation), and they identify mutations in different genes if the diploids can grow at restrictive temperature (complementation).
Explanation:
Recessive mutations are those whose phenotypic effects are only visible in homo-zygous individuals. Moreover, a complementation test is a genetic technique used to determine if two different mutations associated with a phenotype colocalize in the same <em>locus</em> (i.e., they are alleles of the same gene) or affect two different <em>loci</em>. In diploid (2n) organisms, this test is performed by crossing two homo-zygous recessive mutants and then observing whether offspring have the wild-type phenotype. When two different recessive mutations localize in different <em>loci</em>, they can be considered as 'complementary' since the heterozygote condition may rescue the function lost in homo-zygous recessive mutants. In consequence, when two recessive mutations are combined in the same genetic background (i.e., in the same individual) and they produce the same phenotype, it is possible to determine that both mutations are alleles of the same gene/<em>locus</em>.