Answer:
Non-coding DNA.
Explanation:
Inversion is a type of chromosomal abnormality in which the sequence of a segment of a chromosome is inverted or rotated at an angle of 180 degrees. This type of abnormality can change the reading frame of the gene and can cause mutations.
But if the genome sequence is non-coding that is not involved in the formation of protein synthesis than even if the reading frame is inverted will not affect the phenotype of the cell. Also, the non-coding sequences are removed by the splicing mechanisms.
Thus, Non-coding DNA is correct.
Three of the major characteristics used to classify organisms are cell structure, mode of nutrition and cellularity. These characteristics help scientists determine how organisms are similar to each other as well as how they are different from each other.
Answer:
The best explanation if we observe an epithelial cell with chromosomes are visible and two cell nuclei is that the cell has just gone through telophase but not cytokinesis (option b).
Explanation:
A somatic cell, when found in mitosis, exhibits the chromosomes distributed in both poles and the outline of two nuclei in the telophase phase, just before cytokinesis.
In mitotic telophase:
- Chromatids, which are chromosomes, are found in the cell poles.
- It initiates the formation of the nucleus membrane.
- The chromosomes begin to turn into chromatin.
- Disappearance of the mitotic spindle, duplication of organelles and cytoplasmic invagination.
The division and differentiation of the nuclei in telophase is called karyokinesis. Later, cytokinesis occurs, where the daughter cells are separated.
The other options are not correct because:
<em> a and d. In the other phases described, </em><em><u>S and G1,</u></em><em> no chromosome distribution is observed at the poles.</em>
<em> c. A somatic cell does not experience </em><em><u>meiosis</u></em><em>.</em>
Answer:
TCA GCC GGA I think this is right
Answer: <em>D. Causes an accumulation of lipids in brain cells</em>
Explanation:
Tay-Sachs disease is caused by a genetic mutation in the <em>HEXA</em> gene. It is an autosomal recessive disease that causes the mutation on an enzyme, which metabolizes <em>GM2 Ganglioside</em> in nerve cells, this leads to a build-up of the molecule in brain cells. At the moment there is no cure for the disease, only support treatment is available.
