Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
Glycolysis. <span>This is where one 6-carbon molecule of glucose is broken down into two molecules of the three-carbon</span>
Answer: By us people breathing in oxygen and breathing out carbon dioxide.
