Answer:
Please see below
Explanation:
Adapting to the surrounding environment is a critical part of surviving and, eventually, evolution. Take mice as an instance. Their coat color plays a major role in allowing to go undetected from potential predators if they are able to blend in well with the surrounding environment. Mice with a coat color that makes them stand out will be easily preyed on. Hence, those who have <u><em>adapted</em></u> to their environmental conditions will live to pass on their genes. This phenomenon is known as selection pressure.
<span>Rather than being learned, glucose aversion is inherited as an autosomal incompletely dominant trait, which appears to he controlled by
a single major gene. This was discovered through a study done on cockroaches, some were fed regular bait while some were fed bait laced with glucose. Through time they began to avoid the glucose.</span>
An organelle found in large numbers in most cells, in which the biochemical processes of respiration and energy production occur. It has a double membrane, the inner layer being folded inward to form layers (cristae).
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
