Answer:
The best answer to your question: Which type of neuroglia would play a role in controlling glutamate levels in the chemical environment, would be: Astrocytes.
Explanation:
From among the neuroglia, or support cells in the brain, whose purpose is to aid neurons in their different functions, astrocytes are not just one of the most numerous, but also one of the most vital for neuronal support. Amongst one of their most central functions is to help in the control of neurotransmitter emition and retention in the synaptic cleft, between two communicating neurons, and therefore, helps regulate the responses from post-synaptic, and pre-synaptic neurons. It is also responsible for clearing up the presence of ions in the extracellular space, and producing ATP, which regulates the amount of neurotransmitters that are released, and taken, by pre-synaptic, and post-synaptic neurons.
In ALS (Amyotrophic Lateral Sclerosis) the issue with glutamate, a neurotransmitter that excites post-synaptic neurons into releasing excess amounts of calcium, is that this hyper-excitatory response leads neurons, particularly motor neurons, to die, and this is what causes ALS. It has been found through research that astrocytes have to do in this process, but it is not clear yet whether there is a failure in their control system, as ALS is still a condition that is very much under study and still without a cure.
The health care provider suspects the child has developed: Acute post infectious glomerulonephritis.
Acute poststreptococcal glomerulonephritis (APSGN) shows symptoms like infection of the skin or throat and caused by nephritogenic strains of group A beta-hemolytic streptococci. Poststreptococcal glomerulonephritis (GN) is a kidney disease that occurs after infection with certain strains of streptococcal bacteria. The kidneys are responsible for removing waste from the body, regulating electrolyte balance and blood pressure, and stimulating the production of red blood cells. A child is recovering from a bout with group A beta-hemolytic Streptococcus infection. The child returns to the clinic a week later complaining of decrease in urine output with puffiness and edema noted in the face and hands. So these are the symptoms of Acute post infectious glomerulonephritis.
Learn more about Acute post infectious glomerulonephritis on:
brainly.com/question/14684695
#SPJ4
A labeling error i’m pretty sure, if not then a medication error
The application of scientific knowledge for practical purposes, especially in industry. Hope it helps!
