It is because of the different neurotransmitters and their receptors.
Sympathetic stimulation of arterioles in skeletal muscle is mediated by epinephrine acting on its β-adrenergic receptors.
Sympathetic stimulation of vasoconstriction occurs as a result of activation of α-1 adrenergic receptors by norepinephrine released by post-ganglionic sympathetic neurons.
Answer: Purpose
Explanation: If you have no purpose or inclination to have a hypothesis, you wouldn't have a hypothesis in the first place. Plus, it can't be experiment because there isn't anything to experiment without a hypothesis. The analysis is, well analyzing your data to see if your hypothesis is realistic. Definitely, not conclusion. <span />
Answer:
An invasive species
Explanation:
Invasive species come from other environments, many times on accident, but usually thrive so much that they end up taking over and starving other native creatures by taking the resources. Hope this helps! (Brainliest appreciated :))
The correct order is:
- Action potential arrives at the axon terminal.
- Calcium ions enter the axon terminal.
- Synaptic vesicles fuse to membrane of axon terminal.
- Acetylcholine is released into the synaptic cleft.
- Acetylcholine binds to its receptors on the junctional folds.
- Junctional folds become depolarized.
- Action potential is initiated on the sarcolemma.
Action potential travels through the membrane of the presynaptic cell causing the channels permeable to calcium ions to open. Ca2+ flow through the presynaptic membrane and increase the Ca concentration in the cell which will activate proteins attached to vesicles that contain a neurotransmitter (e.g. acetylcholine). Vesicles fuse with the membrane of the presynaptic cell, thereby release their contents into the synaptic cleft-space between the membranes of the pre- and postsynaptic cells. Neurotransmitter binds to its receptors on the postsynaptic membrane and its binding causes depolarization of the target cell (muscle cell).
