If I were you I would choose muscles it seems right to me although I might be wrong. Hope this helped some! ;)
Cell membrane is a semipermable, which means that certain molecules will be able to pass through the membrane. It is contains phospholipids. But for smaller molecules such as CO2 and Water, it can pass through the membrane through simple diffusion. Cell membrane controls the movement of the materials into and out of the cell.
Answer:
The correct insulin pathway is described as under:
2. Binding of insulin to the alpha subunit of the insulin receptor
8. Activation of insulin receptor tyrosine kinase
3. Phosphorylation of IRS proteins
6. Phosphorylation of phosphinositide 3-kinase (PI-3K)
4. Conversion of PIP2 to PIP3
7. Activation of PIP3-dependent protein kinase B (PDK1)
5. Glut4 receptors transported to the cell membrane
Explanation:
The insulin signaling pathway is described as under:
RTK (receptor tyrosine kinases) which is a receptor for insulin is an extracellular receptor but in contrast to other cell surface receptors it is catalytic in nature. In the absence of insulin (ligand), it is monomeric but as soon as it gets activated (activation occurs upon ligand binding), it undergo dimerization. It leads to auto-phosphorylation in it's tyrosine residue which subsequently leads to phosphorylation of tyrosine residue of other receptors. Such hyper-phosphorylated receptor have high affinity with enzyme/molecule like IRS protein which have SH2 domain . IRS down stream activates phosphinositide 3-kinase (PI-3K). This enzyme converts component of animal cell membrane PIP2 into PIP3. PIP3 also remains membrane bound but it has the potential to phosphorylate another enzyme named as PIP3-dependent protein kinase B (PDK1). Further, PDK1 leads to the activation of Akt or PK-B. Akt is a serine-threonine kinase which ultimately leads to the recruitment of Glut4 receptors on cell membrane for uptake of more and more glucose into the cell.
Note: Apart from this Akt also phosphorylates another protein named as FOXO which ultimately causes cell growth, Akt can also phosphorylate BAD protein so as to restrict cell apoptosis or we can say it leads to cell survival, Akt also leads to translation in a cell with the help of mTOR raptor etc.
Answer:
Few crosses
The complexity of cat genetics
Crosses not controlled by the researcher
Explanation:
The purpose of this question is to determine why Megor Grendel is less famous than that of Gregor Mendel.
Gregor Mendel examined pea plants, which have a number of benefits for deducing genetic rules, including:
- The researcher has total control over the crosses.
- Because the peas have both self and cross-fertilization, it is possible to alter the crosses in the simplest way possible.
- Pea plants may be examined for a greater series of generations than cats or other animals.
- Because plant genetics is not overly complicated, several traits may be investigated at the same period.
As a result, the primary factors why Megor Grendel's experiments are not well-known:
- The presence of only a few crossings: It is impossible to establish a genetic theory with such a small number of crossings on the test subject of the organism.
- Cat genetics is too complicated therefore, the fur gene color on the X-chromosome, a characteristics mosaic inheritance. As a result, It is much too complicated to deduce an inheritance pattern.
- Crossings that the researcher cannot fully control. Unlike plants, crosses in animals cannot be totally controlled by the researcher.
As a result, it is impossible to draw any conclusions from them.
A class of lipids called prostaglandins is typically produced at the sites of injury or infection and is used to treat both disease and injury. They regulate functions like blood flow, blood clot formation, inflammation, as well as labor induction.
Arachidonic acid, from whom prostaglandins are generated, is what causes inflammation:
- In synthetic form, prostaglandins are used to induce labor and give birth.
- They trigger uterine contractions and have a ring structure with at least two or three and perhaps more carbons.
Effects of prostaglandins in the body are
- The hormone that circulates and regulates the kidney's reabsorption of sodium. Prostaglandins, which seem to be present in high concentrations, directly affect nerve terminals to induce pain.
- The production of the inflammatory response is significantly influenced by prostaglandins. Their production is markedly increased in inflamed tissue, therefore they help to produce the essential symptoms of acute inflammation.
- NSAIDs (which inhibit cyclooxygenase) as well as COX-2 selective inhibitors, sometimes known as coxibs, were examples of prostaglandin antagonists. Cyclopentenone prostaglandins as well as corticosteroids, which block the formation of phospholipase A2, might help to reduce inflammation.
To know more about Prostaglandins here
brainly.com/question/27180318
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