Answer:
The correct answer is B.
Explanation:
During the follicular phase (first half of female cycle), follicles in the ovary begin developing under the<u> influence of </u><u>FSH.</u> <em>The follicle that acquires more FSH receptors will become </em><em>dominant</em> and will produce more estrogen and inhibin hormone than the others. Inhibin will reduce FSH level and as a result the other follicles will fail to keep growing. At this stage the dominant follicle will become FSH independent.
Estrogen produced by the dominant follicle will stimulate LH secretion. After approximately 24-36 hours from when LH reaches its peak level, the dominant follicle releases an ovocyte. <em>This event is called ovulation.</em>
A. Midline 2 to 3 cm (0.8 to 1.2 in) above the symphysis pubis. Normal ultrasounds use sound waves to produce images, but unlike Doppler devices, they are unable to show blood flow.
The Doppler device ultrasonography is a non-invasive diagnostic that gauges the blood flow through your arteries by reflecting high-frequency sound waves off circulating red blood cells. Doppler ultrasound uses sound waves to monitor artery blood flow. It is used to examine the blood flow to the placenta, uterus, and unborn child throughout pregnancy.It has advantages when used in high-risk pregnancies where the health of the unborn child is a worry. Nowadays, most pregnant women have an ultrasound before 12 weeks. Typically, a fetal Doppler test is performed between weeks 13 and 28 of your second trimester.
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Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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