The nurse is caring for a patient prescribed enoxaparin. The nurse should monitor Complete Blood Count (CBC) and basic metabolic panel (BMP), prothrombin time (PT).
Enoxaparin is low molecular weight heparin (LMWH) and it is used to treat and prevent clinical conditions such as acute coronary syndromes, pulmonary embolism (PE), deep venous thrombosis (DVT), venous thromboembolism (VTE) treatment, and periprocedural anticoagulation.
Enoxaparin has the identical side effects as heparin. Since antidote such as protamine has reduced effectiveness, bleeding complications can be fatal and life-threatening.
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Answer:
The correct answer is option A. False.
Explanation:
John Scott Haldane described a property of hemoglobin which is termed as the Haldane effect. Displacement of carbon dioxide from the hemoglobin of blood by oxygen in the lungs. It promotes the removal of CO₂ from the blood. This process is the Haldane effect.
Decrease in the affinity of the oxygen to binding with hemoglobin in response to a decrease in blood pH due to increased CO₂ concentration in the blood. It is the Bohr effect.
Thus, the correct answer is option A. false.
MCV = Hct × 10/RBC (84-96 fL) •Mean corpuscular Hb (MCH) = Hb × 10/RBC (26-36 pg) •Mean corpuscular Hb concentration (MCHC) = Hb × 10/Hct (32-36%) A rapid method of determining whether cellular indices are normocytic and normochromic is to multiply the RBC and Hb by 3.
Answer:
a. I olfactory
Explanation:
Journey of the Olfactory nerve:
-Originates on the caudal surface of the olfactory bulb
-Crosses the cribriform plate of the ethmoid bone from one part of the critlal galli to the other
-Reachers the olfactory region of nasal cavity
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
