16; D. all of the above
explanation; the gluteus maximus works with the semitendinosus and semimembranosus muscles, extending the thigh. it is, indeed, located in the buttocks, and can be used as an injection site.
17; D. both B and C
explanation; the latissimus dorsi collaborates with the teres major and pectoralis major to adduct and medially rotate the arm at it’s glenohumeral joint.
Change the environment's temperature or move the patient somewhere warmer. Dry off the patient and the bed sheets.
If the temperature is 32.8°C (91°F). External cooling devices or cold normal saline infusions are utilized to quickly reduce body temperature to 89.6° to 93.2° F (32° to 34° C) when therapeutic hypothermia is used postresuscitation.
What is therapeutic hypothermia ?
- Treatments to lower body temperature include therapeutic hypothermia. This lessens injuries and ongoing issues. It is occasionally applied to patients who experience cardiac arrest. When the heart abruptly stops beating, cardiac arrest occurs.
- The difficulties brought on by hypothermia are intended to be avoided through induced hypothermia. It is mostly utilized in neonatal encephalopathy, head injuries, and survivors of cardiac arrest who are comatose. The prevention of cerebral reperfusion injury is suggested to be a mediator for the mechanism of action.
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Answer:
What part of the intra-oral camera is placed in the patient's mouth?
a. Adaptor
b. Wand
c. Pointer
d. Film
Explanation:
Intraoral cameras <em>allow the dentist to explain to the patient his diagnosis in a more direct way, will be of greater impact and will be more involved with the care of his mouth.
</em>
The pointer with the light, is the one that is introduced into the patient's mouth.
<u><em>
The answer is</em></u>: <u>c. Pointer.</u>
Answer:
<u>Ethanol prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u>
Explanation:
Ethylene glycol is an organic compound usually used in antifreeze solutions, solvents and cleaners.
It is abused during self-destruction and accidental intakes.
<u>In the body, ethylene glycol is acted upon by alcohol dehydrogenase and is converted into glycolate and oxalate.</u>
Glycolate and oxalate are both nephrotoxic/ kidney damaging substances. Oxalate precipitates calcium oxalate stones in the kidney. Ethylene glycol poisoning also causes high anion gap metabolic acidosis.
In order to prevent ethylene glycol poisoning, the patient is infused with ethanol, ethanol <u>prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u> In this way, ethylene glycol is not metabolized and the formation of nephrotoxic substances is prevented. Alcohol dehydrogenase instead reacts with ethanol to form acetaldehyde.
