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Answer: pathogen–host coevolution
Explanation:
A major driver of evolution is Reciprocal coevolution between host and pathogen. Rather than pathogen, one-sided adaptation to a nonchanging host, high virulence specifically favoured during pathogen–host coevolution. In all of the independent replicate populations under coevolution, the pathogen ( B. thuringiensis ) genotype BT-679 with known nematocidal toxin genes of C. elegans and high virulence specifically swept to fixation but only some of them go under one-sided adaptation,
so relative change in B. thuringiensis virulence was greater than the relative change in C. elegans resistance is due to the elevated copy numbers of the plasmid containing the nematocidal toxin genes
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Double fertilization-plants with seeds (flowering plants), two male gametes joining with female gametophyte
Gametophyte generation-both (haploid, sexual stage stage-gametophyte, and the diploid stage that produces spores – sporophyte)
Endosperm formed-plants with seed because it is a tissue formed inside the seed which surrounds the embryo and provides nutrition
Mitosis-both (mitosis occurres in spores)
Spores develop into gametophytes-both but, in seedless plants sporophyte produces spores that will develop into a new organism (multicellular gametophyte) using mitosis, while spores of seed plants are produced internally and develop into more complex structures.
It is a reduction of number of cells of their parent cell in half equal numbers.