Answer:
Decomposers get nutrients and energy by breaking down dead organisms and animal wastes. Through this process, decomposers release nutrients, such as carbon and nitrogen, back into the environment. These nutrients are recycled back into the ecosystem so that the producers can use them
Explanation:
a feedback mechanism that maintains homeostasis
Explanation:
Anhidorosis mostly directly interferes with a feedback mechanism that interferes directly with a feedback mechanism that maintains homeostasis.
Since anhidorosis is the inability to properly or normally sweat, it affects the process of homeostasis.
Homeostasis is an important characteristics of life.
It is the ability of organisms to maintain and sustain a balance environment within and outside of them.
Sweating is one such process by which the body carries out homeostasis. On a hot day, the body produces sweat which evaporates and release latent heat to cool the body. This inability affects the intricate balance between the environment and the body.
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Micro molecules all contain oxygen and hydrogen Cho Cho Chon c-carbon h-hydrogen o-oxygen cho cho chon is carbs fats proteins
Gray cannot blend. Dark rocks let black fur blend in. White sones can blend with white fur. Gray would stick out and then not survive. Population of black and white would go up, and then the gray would go down.
Answer:
a. PK2 activates PK1
b. no response would be produced
Explanation:
Kinases are enzymes known to phosphorylate different substrates, thereby activating/inactivating them. In this case, a mutation that produces a constitutively (permanently) active PK1 kinase is itself able to continue the signaling pathway, independently of whether or not PK2 is present. This constitutive mutation shows that PK1 is recruited to continue signaling events within the cell and, therefore, PK1 activation is downstream in the signaling pathway. Conversely, cells containing active PK2 and inactive PK1 would be unable to continue the signaling pathway since PK2 activation is upstream of the induction of PK1, and thereby these cells cannot respond to the signal (PK2 activation).
