Answer:
Sleepytime medican and her mother
Explanation:
a nurse is teaching a group of clients about risk factors for heart disease. The nurse include that Obesity, Hypertension increase a client's risk for a myocardial infarction (mi).
<h3>What is
myocardial infarction ?</h3>
The heart muscle starts to die as a result of inadequate blood flow during a heart attack, which is medically referred to as a myocardial infarction. Typically, a blockage in the arteries supplying blood to your heart is what causes this.
<h3>Causes of
myocardial infarction :</h3>
The primary cause of death in the US, coronary artery disease, is to blame for the majority of myocardial infarctions. The myocardium is deprived of oxygen when coronary arteries are blocked. Myocardial cell loss and necrosis can result from a prolonged lack of oxygen flow to the myocardium.
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Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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Answer:
( a ) Voltage gated channels - channel can open and close based on membrane potential. At rest, the channel is closed and it opens at threshold.
Explanation:
Voltage Gated Channels -
It is a channel with a cluster of proteins present in the membrane .
The function of voltage - gated channel is to open for the some values of membrane potential and close for others . In maximum cases , the relation between channel state and membrane potential is probabilistic and requires a time lag . The ion channel can even flip the conformations at an unpredictable times , the membrane potential even determines the rate of transition and the probability per unit time of each and every transition .
Answer:
La salud sexual es un estado de bienestar físico, mental y social en relación con la sexualidad. Requiere un enfoque positivo y respetuoso de la sexualidad y de las relaciones sexuales, así como la posibilidad de tener experiencias sexuales placenteras y seguras, libres de toda coacción, discriminación y violencia.
Explanation: