According to the research, the correct option is Period of invasion. The stage of an infectious disease when specific signs and symptoms are seen and the pathogen is at peak activity is the Period of invasion.
<h3>What is an infectious disease?</h3>
It is the disease that is due to the invasion of the body by pathogenic germs, which establish and multiply caused by a local cell injury, secretion of toxins or by the antigen-antibody reaction.
The stage of an infectious disease called the Period of invasion is the time interval between invasion by an infectious agent and the appearance of the first signs and symptoms of the disease.
Therefore, we can conclude that according to the research, the correct option is Period of invasion. The stage of an infectious disease when specific signs and symptoms are seen and the pathogen is at peak activity is the Period of invasion.
Learn more about stages of an infectious disease here: brainly.com/question/14454614
#SPJ1
This could have been a case of polymedication, but I doubt it since it is an 82-year-old woman. I'm not sure if they are antibiotics, but benzodiazepines and anticholinergic drugs can cause amnesia. Or, it is a possibility her primary care doctor gave her the wrong dosage of the antibiotics, causing side effects, one of them possibly being amnesia.
Hybridomas, which produce monoclonal antibodies, are made by fusing cells of the immune system with B lymphocytes and myeloma cells.
<h3>
What are Hybridomas?</h3>
- Large-scale production of monoclonal antibodies is made possible through hybridoma technology.
- An antigen that triggers an immune response is first injected into a mammal to begin the process.
- A specific sort of white blood cell called a B cell makes antibodies that bind to the antigen that has been injected.
- These antibody-producing B-cells are then removed from the animal and combined with immortal B cell cancer cells, or myeloma, to create a hybrid cell line known as a hybridoma.
- It possesses both longevity and procreative capacity of the myeloma and the antibody-producing capacity of the B-cell.
Hence, the creation of hybridomas, which result in the production of monoclonal antibodies, involves fusing immune system cells with B lymphocytes and myeloma cells.
To learn more about Hybridomas refer to:
brainly.com/question/15189667
#SPJ4
Answer: Mutations in the GALT, GALK1, and GALE genes cause galactosemia.
Explanation: Galactosemia is a disorder that affects how the body processes a simple sugar called galactose. A small amount of galactose is present in many foods. It is primarily part of a larger sugar called lactose, which is found in all dairy products and many baby formulas. The signs and symptoms of galactosemia result from an inability to use galactose to produce energy.Classic galactosemia, also known as type I, is the most common and most severe form of the condition. If infants with classic galactosemia are not treated promptly with a low-galactose diet, life-threatening complications appear within a few days after birth. Affected infants typically develop feeding difficulties, a lack of energy (lethargy), a failure to gain weight and grow as expected (failure to thrive), yellowing of the skin and whites of the eyes (jaundice), liver damage, and abnormal bleeding. Other serious complications of this condition can include overwhelming bacterial infections (sepsis) and shock. Affected children are also at increased risk of delayed development, clouding of the lens of the eye (cataract), speech difficulties, and intellectual disability. Females with classic galactosemia may develop reproductive problems caused by an early loss of function of the ovaries (premature ovarian insufficiency). Galactosemia type II (also called galactokinase deficiency) and type III (also called galactose epimerase deficiency) cause different patterns of signs and symptoms. Galactosemia type II causes fewer medical problems than the classic type. Affected infants develop cataracts but otherwise experience few long-term complications. The signs and symptoms of galactosemia type III vary from mild to severe and can include cataracts, delayed growth and development, intellectual disability, liver disease, and kidney problems.
