I am not sure how to answer
<span>sulfuric acid - acid rain. Damages forests, limestone statues/buildings, lakes (alters pH) etc. Sulphur trioxide is a pollutant, which is also detrimental to health. Mark me the brainiest!</span>
Answer:
Conduction
Explanation:
convection- not related, as it is hot air rising and cold air falling, which isn't happening.
Subduction- that has to do with the Earth's plates moving
Radiation- doesn't have to do with heated substances.
Answer:
I wasn't quite sure what virus you were referring to in your question, but here's a general answer: Viruses use their host cells' machinery to replicate themselves.
If they are a specific type of virus known as a retrovirus, they have the ability to use the host cells' enzymes to change the RNA contained within the virus into DNA (via some type of replication I suppose).
In other cases, if they contain DNA instead of RNA (that is, the virus), they can use the host cell's machinery to create RNA via enzymes involved in transcription and/or they can incorporate that DNA into the host cell's DNA. This is part of a type of viral replication cycle known as the lysogenic cycle.
In another type of viral replication cycle known as the lytic cycle, the virus simply has itself and its genome duplicated until the host cell bursts, releasing the viral material. Here, again, the virus uses the host cell's machinery to replicate itself.
Answer:
The options
A)Damage to cellular mitochondria
B)Increased ATP levels
C)Activation of the p53 protein
D)Apoptosis
The CORRECT ANSWER IS D
D)Apoptosis
Explanation:
The extrinsic pathway of apoptosis includes extracellular signaling proteins which adhere to cell surface molecules known aa death receptors which in turn activates apoptosis.
The aftermath activates endonucleases prompting division of DNA and ultimately cell death.
Apart from the TNF and Fas ligand, primary signaling molecules also promotes the extrinsic pathway, examples of such are the TNF-related apoptosis-inducing ligand (TRAIL); the cytokineinterleukin-1 (IL-1); and lipopolysaccharide (LPS), the endotoxin located in the outer cell membrane of gram-negative bacteria while the activation of the p53 protein, and decreased ATP levels in the intrinsic pathway results in DNA damage.