Answer: Yes, it is unusual to have prominent symptoms of ADHD before age of two and almost many adults were never been diagnosed about ADHD in their childhood. Among them most of the people outgrow the symptoms but some of them develops a range of new symptoms in adolescent and adult stage of life. ADHD is worldwide prevalent in Children of 3 to 6 years of age with percentile of 5.29.
Explanation: ADHD, Attention Deficit Hyperactivity Disorder main cause is not exactly known but it is mainly a neurotransmitter or dopamine dysregulation at the early stage of life also research have proven that it runs down in a family. Apart from inheritance or Genetic factors, Pregnancy problem like low birth weight, exposure to alcohol smoke lead might become the factors to impose ADHD to individuals. ADHD symptoms likely to appear either as early in the age of 3 or before the age of 7. The symptoms might continue to adolescent or adult stages or often go away. The symptoms are probably different for every age group. Like Children display inattentive behavior, easy to distract from external stimuli, learning disabilities, unable to solve school work, low self-esteem, emotional dysregulation, low frequency of indoor and outdoor activities and anxiety. If the symptoms advances to adolescent or adult stage the person would suffer challenges in decision making, planning and organizing, maintaining relationship with parents and sibling and also in making friends. The ADHD can be treated by Behavior therapy and parents training and educational programs.
Answer:
i tnk its um no thats not it um its the first
Explanation:
Opportunistic infections (OIs) are infections that occur more frequently and are more severe in individuals with weakened immune systems, including people with HIV. People are at greatest risk for OIs when their CD4 count falls below 200, but you can get some OIs when your CD4 count is below 500.
1. Patent means open.
2. Appear in the groin.
3. Symptom of Gastritis (might wanna double check this one)
Answer:
Altogether, BPA accumulation in eggs, mimicking maternal transfer, affects larval growth and the mode of action involves disruption of genes involved in the GH/IGF and thyroid axes function in trout.
