Answer:
Cell’s who make errors when dividing further their risk for becoming cancerous
Explanation:
During cell division, DNA is replicated. If a mutation occurs during replication that cannot be repaired a cell will undergo apoptosis (programmed cell death). Sometimes, mutations occur to tumor suppressor genes and proto-oncogenes. These genes make sure a cell has no mutations before dividing/making the decision to start apoptosis. When these genes become mutated, the cell is less likely to correct potential mutations and is more at risk for becoming cancerous.
I think the answer is D.) making a fully competent patient receive a treatment, even if they don’t want the treatment
Answer:
If your quality of sleep is being disturbed. And when you start to have a rash... then I would see a doctor.
Explanation:
Answer:
When there is a malfunction of the cell reproduction sequence regardless of the cause (whether it is damaged by mutation, internal or external disruption of the cell reproduction cycle or other factors, the most approximate result is Cancerous growth.
Explanation:
The genes which regulate cell growth can be likened to the brakes of a vehicle. The brakes ensure the car stops when it has to. This prevents accidents, damages, death and destruction of property.
When the mechanism for the replication and replacement of cells go out of control, all sorts of malformations emerge. We term them cancerous tissues.
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Since the hemagglutinin gene is how a virus is able to get into a cell, and the neuraminidase gene disperses those copies into the body. In order to have it transferred from human to human is for the hemagglutinin gene to change.
