Answer: Sulfa antibiotics target a specific enzyme that inhibits bacterial growth. ... Viruses are unaffected by antibiotics because they do not have peptidoglycan cell walls or ribosomes, and they do not replicate their own DNA. Bacteria can become resistant to antibiotics through the process of selection and evolution.
Explanation:
EEF or electroencephalogram expensive and time consuming.
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Answer: C, If a population is spread out over too large area, they may become extinct.
In the presence of cAMP, the R subunit is bound by four molecules of cAMP, allowing the catalytic subunits to dissociate, translocate to the nucleus, and then phosphorylate CREB. CREB then homodimerizes, binds to the CRE (cAMP response element) in the promoter of target genes, and activates transcription. Drugs could be designed that would interrupt signaling anywhere along the pathway. For example, a drug could stabilize the R and C subunit interactions of PKA and block translation into the nucleus. A drug could prevent interaction of cAMP with the R subunit and essentially lead to the same result. Similarly, a drug could have a more downstream action that might prevent phosphorylation of CREB, thus not allowing it to become activated. Another option would be to prevent the required dimerization of CREB, thus preventing its ability to bind DNA. Hope this helped ;)
