Answer:
The answer is 3. Double Helix.
Answer:
The mutation is on chromosome/karyotype 8.
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Following spdf notation, the electron configuration for Nobelium (No) is 1s2 2s2 2p6 3s2 3p6 3d10 4s2 4p6 4d10 5s2 5p6 4f14 5d10 6s2 6p6 5f14 6d0 7s2
Intracellular Potassium Shifts and Impaired Potassium Excretion leads to elevated plasma K levels.
What is Hyperkalemia?
- Hyperkalemia, a potentially lethal condition, develops when serum potassium levels rise above 5.5 mmol/l.
- Potassium is the most common intracellular cation and is crucial for many physiological functions, at a concentration of 100-150 mmol/l. The digestive system quickly and usually fully absorbs potassium.
Causes:
- Increased Potassium intake: In adult patients with normal renal function, increased dietary potassium intake is a very rare cause of hyperkalemia, but it can be a significant factor in people with kidney disease.
- Intracellular Potassium Shifts: Large amounts of intracellular potassium can be released into the extracellular area as a result of cellular damage. Excessive activity, rhabdomyolysis after a crush injury, or other hemolytic processes can all be to responsible for this.
- Impaired Potassium Excretion: The most frequent cause of hyperkalemia is acute or chronic renal disease. Hyperkalemia may also result from tubular dysfunction brought on by aldosterone insufficiency or insensitivity.
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