Answer:
Women who carry one copy of the mutated gene still have normal color vision because they have another copy, which is not mutated, in the other X chromosome that will be the dominant one. As a result, the women are carriers of the mutated gene but not color blind.
Explanation:
Colorblindness is a sex-linked mutation. A woman has two X chromosomes, while a man has one X chromosome and one Y chromosome. The mutated gene that causes color blindness is on the X chromosome, and it is OPN1LW. So if a woman has one mutated OPN1LW in one of her two X chromosomes, the OPN1LW gene in the other X chromosome will be the dominant one stopping the woman from being colorblind.
In the case of men, as they only have one X chromosome, if there is a mutation on the OPN1LW in the X chromosome, the men will be colorblind because there is no extra copy of the gene, as it is in women.
Answer:
The dispersion pattern of the population depends on the type of the population and their distribution in the environment. Different types of dispersion are uniform, random and clumped.
The uniform dispersion occur when the population is evenly spaced out in the area. Random dispersion is independent of the other individuals and randomly spaced. Clumped dispersion is greatly influenced by the behavior and the resources. Population are present in small patches.
Answer:
Nucleotides are organic molecules consisting of a nucleoside and a phosphate. They serve as monomeric units of the nucleic acid polymers deoxyribonucleic acid (DNA) and ribonucleic acid (RNA), both of which are essential biomolecules within all life-forms on Earth.
Explanation:
Answer:
Explanation:
The higher temperature is the factor which is responsible for the process of denaturation of proteins in which the breaking down of hydrogen, disulphide bonds and destabilization occurs. This process breaks up the bond between the polypeptides.
The bovine pancreatic trypsin inhibitor comprises of 58 amino acids this consists of the disulphide bonds which are destroyed on heating as a result of this the inhibitor becomes inactive. But on cooling it comes to the original form as the internal structure starts making hydrogen and disulphide bonds again. Thus the activity of the BPTI again gets restored.
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