In response to a notification of a <u>decrease</u> in blood pH by the nervous system, the brain sends signals to the external intercostal muscles and the diaphragm.
<h3>What is
blood pH?</h3>
Blood pH can be defined as a measure of the molar concentration of hydrogen ions that are present in the blood of a living organism, with respect to its acidity, neutrality or alkanlity (basicity).
In response to a notification of a <u>decrease</u> in blood pH by the nervous system, the brain would send signals (impulses) to the external intercostal muscles and the diaphragm through its respiratory center, so as to help the living organism increase its breathing rate and the volume of its lungs during inhalation.
Read more on blood pH here: brainly.com/question/11209525
Lamarck called his theory, the theory of Acquired Characteristics. Lamarck's Theory of Inheritance of Acquired Characteristics has been disproved. This was done in two major ways. The first is by experiment. We have seen through many real examples and observations that changes that occur in an animal during life are not passed on to the animal's offspring.
C. crossing over This is because each gene have a piece of each other Hope this helped=)
Answer is Plants and animals both break a phosphate bond of ATP to release energy.
In both plants and animals adenosine triphosphate (ATP) is the main molecule for storing and transferring energy in cells. It is also called the energy currency of the cell. ATP molecule composed of three phosphate groups. These phosphate groups are linked to one another by two high-energy phosphoanhydride bonds. When energy is required by the cell, one a phosphoanhydride is bond broken removing one phosphate. As a result energy is released and ATP is converted to adenosine diphosphate (ADP).
Answer:
Indivisuals with producing an abnormal form of extracellular protein fibrillin are suffering from Marfan syndrome ehich is caused by genetic mutation in the FBN1 gene.
Explanation:
Gene mutations in FBN1 gene results in the production of an abnormal extracellular matrix fibrillin-1 protein that cannot function properly. These gene mutations basically reduce the amount of fibrillin-1 produced by the cell, alter the structure of fibrillin-1, or causes the impairment of the transport of fibrillin-1 out of the cell.
As a result, protein is poorly incorporated into extracellular matrix. Hence, indivisuals with Marfan syndrome present following symptoms
Tall stature.
Disproportionately long arms, legs and fingers.
Sternum either protrudes outward or dips inward.
Arched palate and crowded teeth.
Heart murmurs.
Extreme nearsightedness.
