Answer:
commensalism mutualism,parasitism
The extra X chromosome is received from mother.
<h3><u>Explanation</u>:</h3>
Klinefelter's syndrome is the condition of chromosomal aberration where the person has an extra X chromosome. This means the genotype of the person is 44+XXY and the total chromosome number is 47. This condition is achieved due to inappropriate meiotic division and one of the sperm or ovum has the an extra X chromosome.
Here both the parents are normal in terms of colour vision. So the father cannot have the gene of colour blindness because if it was present, then father would have been colour blind because he has only one X chromosome per cell. So its present in mother who is heterozygous in terms of colour blind gene. So the extra X chromosome also came from her which made the presence of two defective X chromosomes in the offspring and made it colour blind.
The answer for this is true :)
Answer:
they form covalent bonds
Explanation:
jeesh dude j answer the dammn question
Answer:
All of these choices are correct.
Explanation:
Cell cycle is the process of growth and division of cell. It comprises of interphase and mitosis. In interphase the cell grows, replicates its genomic content and prepares itself for division. In mitosis the division occurs.
Cell cycle is controlled by a group of kinases called as Cyclin dependent Kinases (CDKs). They act by phosphorylating their substrates. They are of various types like Cdk1, Cdk2, Cdk4 etc. They become active when they bind to a regulatory protein called cyclin. They are also of various types like Cyclin A, Cyclin B, Cyclin C etc. Level of cyclin and corresponding CDK increases and decreases according to the stage of cell cycle. For example in S phase of cell cycle concentration of cyclin A and E shoots up. CDK2 is able to bind to these cyclin molecules and hence it becomes active.
Cell cycle has major checkpoints where the condition of cell is analysed before it proceeds to the next stage of cycle. If any abnormality is detected, repair mechanism is activated or the cell is killed. Checkpoints do not allow cell cycle to proceed in damaged cells.
p53 is a tumor suppressor protein which can halt cell cycle when it detects some abnormality in cell. It usually acts in G1/S checkpoint (before the DNA replication starts in cell) and G2/M checkpoint (before the cell division begins). Hence, all of the above statements are true.
