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Rainbow [258]
3 years ago
9

What did Mendel's cross pollination of pea plants prove

Biology
2 answers:
DaniilM [7]3 years ago
5 0
I'm pretty sure that the answer is D.
pashok25 [27]3 years ago
4 0

Answer: The correct answer is- D certain factors are dominant and other factors are recessive.

Mendel was interested in knowing the offspring that is produced from the cross pollination (that is from two different plants) as he wanted to study different traits of pea plant.

For doing so, he first removed the anthers of one plant (to prevent self pollination) and then transferred the pollens of one plant into the stigma of another plant.

When he allowed the cross pollination of purebred pea plant with other plant, he observed that some factors are dominant (such as tall height of pea plant, which masks the expression of recessive factors) and other are recessive (such as dwarf height of pea plant).

Thus, option D) is the right answer.

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A gene on human chromosome 15 is expressed throughout the body. However, in the brain, only the maternal copy of the gene is exp
lord [1]

Answer:

C) Through genomic imprinting, methylation regulates expression of the paternal copy of the gene in the brain.

Explanation:

The pattern of gene expression wherein either paternal or maternal gene is expressed in specific cells while the other one is prevented from expression is known as genomic imprinting.

In the given example, the maternal copy of the gene on chromosome 15 is expressed in brain cells while its paternal copy is not expressed in these cells. Hence, the pattern of expression of this gene is regulated through genome imprinting. One of the mechanism is methylation of cytidine residues of CpG islands of the DNA that are more frequently present within promoters of the genes.

When the cytidine residues of these sequences are methylated into 5-methylcytidine, the transcription factors do not bind to these promoters preventing the expression of these genes.

Hence, methylation of cytidine residue in CpG islands of the promoters of the gene present on chromosome 15 could have silenced its expression in brain cells.

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Explanation:

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