Answer:
(A) It prevents electron flow from the iron-sulfur centers in complex 1 to the ubiquinone. Due to reduction in electron transfer rate, there is a decrease in the production of ATP which is dangerous for some insects and fish over time.
(B) It also prevents electron flow from cytochrome b to cytochrome c1 at the complex III which leads to QH2 accumulation. If oxidized Q is not present, these is alteration of electron flow and the production of ATP is altered.
(C) Rotenone only prevent electron transfer into the chain at Complex 1 but it does not affect electron transfer at Complex II. Although there is slow ETC, it does not stop completely. However, Antimycin A prevents the oxidation of QH2, the final electron acceptor crom complex I and complex II. Thereby, stopping the production of both ETC and ATP. It can be concluded that antimycin A is a more potent poison.
Explanation:
Rotenone prevents electron flow from the iron-sulfur centers in complex 1 to the ubiquinone. Due to a reduction in electron transfer rate, there is a decrease in the production of ATP which is dangerous for some insects and fish over time. Antimycin A also prevents electron flow from cytochrome b to cytochrome c1 at the complex III which leads to QH2 accumulation. If oxidized Q is not present, there is an alteration of electron flow and the production of ATP is altered. Antimycin A is more potent than rotenone.
Monoploid organisms reproduce asexually since they need to transmit all of their genetic material to their offspring. Diploid organisms, have 2 copies of their genetic material that differ slightly in their genes. Since the progeny gets half of the DNA from each parent, we have that new combinations can emerge; for example, if the mother is AA for some allele and the father aa, their offspring will be Aa, a new genotype. This might have different implications (for example, the recessive gene for thalassemia also provides resistance to malaria). Finally, during meiosis, there is also an event called crossover that increases the genetic variation of the offspring.
The phosphorylation of fructose 6-phosphate to fructose-1,6-bisphosphate is the committed step in glycolysis because<u> fructose 1,6-bisphosphate can undergo no other reactions than those of glycolysis.</u>
<h3>
What is phosphorylation?</h3>
- The crucial process of glycolysis involves the breakdown of glucose into two molecules of pyruvate. It involves a number of steps and many enzymes.
- It takes place over the course of ten phases, demonstrating how important and crucial phosphorylation is to the production of the final goods. Step 1 of the preliminary step (first half of glycolysis) and step 6 of the payout phase reactions are started by phosphorylation (second phase of glycolysis).
- Because fructose-6-phosphate cannot cross the cell membrane, it is forced to remain inside the cell. Step 3 involves phosphorylation, when fructose-6-phosphate is changed into fructose-1,6-bisphosphate.
To learn more about phosphorylation with the given link
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The Nucleus is the control center, or brain of the cell.
