Answer:
Hyperglycemia means high blood sugar. The hormone released by the pancreas in response to high blood sugar is insulin.
Explanation:
Hyperglycemia, or high blood sugar (high blood glucose), happens when the body does not have enough insulin or when the body does not use insulin properly, so the level of sugar in the blood is higher than it should be. It is especially dangerous for people with diabetes. In this case, it is because you ate too much carbohydrate in your meal, which causes the blood sugar to go up for a few hours. In response to this, high blood sugar, the pancreas releases insulin, which helps break down the ingested glucose (sugar).
I think you got it right; emotional, physical, personal.
Answer:
You should treat her as a patient and a normal person, as you should treat any patient. Do not let personal connections get in the way of a professional mental disorder situation. Treat her with care and as a patient. At work play your role as coworker, and in the group play the role as caregiver.
Explanation:
Answer:
Multiple sclerosis (MS) is the most common demyelinating and an autoimmune disease of the central nervous system characterized by immune-mediated myelin and axonal damage, and chronic axonal loss attributable to the absence of myelin sheaths. T cell subsets (Th1, Th2, Th17, CD8+, NKT, CD4+CD25+ T regulatory cells) and B cells are involved in this disorder, thus new MS therapies seek damage prevention by resetting multiple components of the immune system. The currently approved therapies are immunoregulatory and reduce the number and rate of lesion formation but are only partially effective. This review summarizes current understanding of the processes at issue: myelination, demyelination and remyelination—with emphasis upon myelin composition/architecture and oligodendrocyte maturation and differentiation. The translational options target oligodendrocyte protection and myelin repair in animal models and assess their relevance in human. Remyelination may be enhanced by signals that promote myelin formation and repair. The crucial question of why remyelination fails is approached is several ways by examining the role in remyelination of available MS medications and avenues being actively pursued to promote remyelination including: (i) cytokine-based immune-intervention (targeting calpain inhibition), (ii) antigen-based immunomodulation (targeting glycolipid-reactive iNKT cells and sphingoid mediated inflammation) and (iii) recombinant monoclonal antibodies-induced remyelination.Keywords: calpain, central nervous system, demyelination, fingolimod, glycolipids, lipids, multiple sclerosis, myelin, myelination, NKT cells, oligodendrocytes, remyelination, T cells
Explanation:
The answer would be, Low self-esteem
