Answer:
There won't be any functional viral DNA synthesis
Explanation:
First of all, you must consider that the HSV has a double-stranded, linear DNA genome. Furthermore, the DNA skeleton is made of the 5'-3' phosphodiester bond. In other words, the 5-phosphat of a nucleotide will bond the 3' OH of the Deoxyribose (see image).
Therefore, if you add a molecule which lacks the 3' OH, such as acyclovir, and has a similar chemical structure to a regular nucleotide, the DNA polimerase will be able to add acyclovir to the main backbone of the viral genome. All in all, you'd be ''cheating'' the DNA pol.
Hence, wherever acyclovir was integrated, there won't be any chance to form a 5'-3' phosphodiester bond. The protomolecule will not be stable and will soon be degradated.
Answer:
Evolution changes the abilities of the Frigate birds.
Explanation:
The Frigate birds have webbed feet because they live in the ocean and unable to fly due to its big weight. Before the evolution, the Frigate birds lives in the ocean due to their large body weight but with the passage of time, evolution occurs that leads to formation of flying ability in the bird so now the bird have the old characteristics of their ancestor as well as new abilities in the physical traits of the Frigate birds due to the evolution.
The first evidence of modern human traits, including increasing brain size and dependence on material culture shows up in Homo habilis. Even though they are species<span> of genus </span>Homo, they are not similar to people nowadays at all. <span>In </span>palaeoanthropology, these species were the first ones who left Africa and started to move and spread along Eurasia, which is one the main facts and characteristics of Homo habilis.
The answer to this question is DNA ONLY!
This statement is true.
In the case of autosomal dominant disease, the person is either homozygous (which is very rare) or heterozygous. On the molecular level, either the mutation produced a new deleterious protein for the organism, or the mutation affected an existing protein in the physiological state and that a 50% activity is not enough to compensate for the needs. of the body.
In case of autosomal recessive disease, the sick person is always homozygous. If the two loci each have a different mutant allele, it is called a "composite heterozygote". People with autosomal recessive inheritance disorder have "mandatory heterozygote" parents.
