Professional antigen-presenting cells, such as phagocytes, are the first cells to encounter a pathogen (apcs). This means they have the ability to activate cells of the adaptive immune system.
<h3>What is meant by Pathogen?</h3>
- A pathogen is an organism that infects its host with disease, and the severity of the disease symptoms is referred to as virulence.
- Pathogens include viruses, bacteria, unicellular and multicellular eukaryotes, and a wide range of other organisms.
- The bone marrow of an average healthy adult generates 100 billion new neutrophils per day. Because there are so many neutrophils in the body at any given time, they are often the first cells to reach the site of an infection.
- Long thought to be a component of the innate immune system, complement has recently emerged as an important regulator of the adaptive immune response.
Therefore the correct option is D) can activate cells of the adaptive immune system.
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Answer:
Its either B or D but i think its B
Explanation:
i hope it B if not then its D
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
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Answer:
A trait can take on almost anything! Like your hair, your eyes, your nose, and... sometimes even the way you speak!
Explanation:
