There were A adults, Sm male students, and Sf female students.
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<span>Sm = 1 + 7A </span>
<span>Sf = Sm/2 </span>
<span>A + Sm + Sf = 82 </span>
<span>A + (1 + 7A) + (1 + 7A)/2 = 82 </span>
<span>2A + 2 + 14A + 1 + 7A = 164 </span>
<span>23A + 3 = 164 </span>
<span>23A = 141 </span>
<span>A = 7 </span>
<span>The number of male students is 50. </span>
<span>The number of female students is 25.</span>
Answer:
It would be the plant type
Explanation:
This is because you are measuring the growth of 2 different plants, so they can not be the same. all the other options ARE constants
A. If the prey isn’t getting killed, they will continue to increase the population until they hit their max (due to resources).
Today, any environment surrounded by other ecosystems that are unlike it is subject to Wilson’s theory of island biogeography. Because they are geographically isolated from other related ecosystems, these ecologies are referred to as "islands." Waterbodies divide tropical islands, but this idea also takes into account mountaintops, caverns, and other isolated ecosystems.
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What is Wilson’s theory of island biogeography?</h3>
- The biologist Edward O. Wilson and environmentalist Robert MacArthur published The Theory of Island Biogeography in 1967. It is widely considered as a foundational work in the ecology and biogeography of islands. The book was reissued by the Princeton University Press in 2001 as a volume in their "Princeton Landmarks in Biology" series.
- The hypothesis that insular biota maintain a dynamic equilibrium between extinction and immigration rates was made more well-known by the book. An island's pace of new species immigration will decline as the number of species increases, while the rate of extinction of native species will rise.
- Thus, MacArthur and Wilson anticipate that there will come a point of equilibrium where the rate of immigration and the rate of extinction are equal.
To learn more about The Theory of Island Biogeography with the given link
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Such changes would occur mostly likely near or in the active binding site of the enzyme.
Because the drugs used are competitive inhibitors of the <span>HIV reverse transcriptase enzyme, it means that they connect directly to the active binding site of this enzyme not allowing it to preform its function. If the mutations impede this drugs to work, it is probably because they alter the active binding site of the enzyme, not allowing the drug to bind and have its competitive behaviour permitting the enzyme to work normally. </span><span /><span>
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