Given the answers;
a) Reptiles
b) Cactus plants
c) Small birds
d) Crabs and crayfish
The answer is C. Small birds.
On his visit to the Galapagos Islands, Charles Darwin discovered several species of finches that varied from island to Island, which helped him to develop his theory of natural selection.
DNA, ribosomes, cytoplasm
The plato answer is:
Celiac disease damages the intestinal villi. As a result, nutrients pass through the digestive tract without being broken down and absorbed properly. This indigestion will eventually cause irritation in the intestine as well as gastrointestinal discomfort.
<h2>Biological predispositions</h2>
Explanation:
- Biological predisposition is when a subject (human, animal, plant) possesses some internal quality that gives them an increased likelihood of having a condition. This is a technical definition, and what it's saying is this living thing has a higher probability to have condition X, Y, or Z. The title of the section alludes to a quality in something's past that will influence its future
- Genes are molecular units for trait heritability. They're often made up of groups of DNA. DNA makes proteins, proteins make cells, cells make tissues, tissues make organs, and organs make up you. Many people like to point out that humans share 98% of their genes with chimpanzees. While this is mostly true, it should also be noted that humans share about 60% of the same DNA with banana plants. This means that a great deal of what genes do is fundamental, such as how to build cell walls and convert sugar to energy
- Hence, the right answer is "biological predispositions"
Answer:
The correct answer is ''loss of acetylcholine receptors in the end-plate membrane.''
Explanation:
Myasthenia gravis represents a human autoimmune disease. The neuromuscular junction abnormalities that occur in this disease are due to antibody-mediated processes. A reduction in the number of functional acetylcholine receptors on the postsynaptic membrane was established to contribute to the physiological abnormalities of myasthenia gravis. The reduction in receptors is due to immunological alterations directed against the acetylcholine receptor. Normally, when acetylcholine binds to its receptor, it opens allowing the rapid entry of cations, especially sodium. With this, the depolarization of the postsynaptic region of the muscle fiber occurs. Although acetylcholine is normally released in myasthenia gravis, it is not capable of causing an action potential in the muscle sufficient to initiate or weaken contraction. In patients with myasthenia gravis, failure to activate muscles is responsible for myasthenic fatigue. Most patients have elevated serum titers of antireceptor antibodies, which in turn accelerate receptor degradation.
