Something that requires a lot of energy, such as active transport, as more mitochondria means more respiration.
Answer:
combustion
Explanation:
glucose is being burnt by air (oxygen) to produce carbon dioxide and water
The answer is false because it's false
Answer;
A Both make it possible for the conditions in the interior to be different from the conditions outside.
Explanation;
Groups of molecules called microspheres may have preceded the living cells of today. Microspheres are tiny bubbles filled with groups of large organic molecules; they can form under very specific conditions. These microspheres may be a lot like the vesicles formed from the organic compounds taken off of the modern meteorites. Microspheres are not cells, but they do share some characteristics with cells.
These bundles of molecules are able to maintain an internal environment different from the surroundings outside the bubble. They also have a simple way of storing and releasing energy. They expand by absorbing additional molecules until they reach an unstable size, and then they split into smaller microspheres. This division is not true reproduction or cell division, but it may be a precursor to it.
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
