Answer:
the plant produces essential oils in small glands around the foliage and flowers.
Explanation:
Answer:
Cells duplicate and condense their DNA prior to entering mitosis. ... Early microscopists were the first to observe these structures, and they also noted the ... forms of chromosomes leading up to and during cytokinesis, the ultimate division of one ... condensin forms rings that coil the chromosomes into highly compact forms.
Explanation:
Answer:
The correct answer would be option D) no gametes.
Explanation:
The ABC model of the flower was formulated to describe the collection of genetic procedures that determines floral organs in several plants. These species consist of four whorls which are sepals, petals, stamens, and carpels. These whorls are expressed by the expression of a number of genes present in each whorl or verticli.
Gene A is solely responsible for the sepals while the co-expression of A and B genes are responsible for the petals. The identity of the stamens is expressed by B and C and the carpels require C and D genes to be active.
The homozygousity for a normal mutation in gene C will lead to inactive or lack stamens and carpels, which are male and female reproductive organs. These are an important structure for forming gametes.
Thus, the correct answer would be option D) no gametes.
True due to the hormones that act upon the kidneys Two examples being Antidiuretic Hormone (ADH) and Aldosterone
ADH increases water permeability of the kidney tubules and Aldosterone decreases the Sodium in the urine and increases the Potassium.
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
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