Answer:
The correct answer is option D. give furosemide.
Explanation:
Digoxin is a drug that uses in the cardiovascular condition. It inhibits the Na-K ATPase enzyme, which functions to maintain the intracellular environment by regulating the entry and exit of sodium and potassium.
Digoxin toxicity may occur due to the low level of potassium (inside the cell)due to the inhibitaion of the sodium-potassium pump. Furosemide is a diuretic drug that decreases salt concentration (such as ions) in the body by increasing urine output. and causes a low level of potassium in the body
Thus, furosemide increases digoxin toxicity as during low levels of potassium, digoxin can easily bind with the sodium-potassium pump and shows severe effects.
Put on clean clothes after washing with soap and water. Make a call to the authorities and go to the hospital. It can be suggested that you isolate yourself or stay away from other people. If your symptoms are similar to those listed and you fall into the category of those who are at risk, get emergency medical help right once.
<h3>How are bioterrorists handled?</h3>
The CDC recommends intravenous ciprofloxacin or doxycycline together with one or two other antimicrobials, as indicated above, to treat anthrax inhalation in a bioterrorism scenario. Therapy can be altered further when additional data on antibiotic susceptibility becomes available and the clinical situation permits.
Supportive care and antitoxin are included in the treatment of severe symptoms. Biochemical Discord: It's a neurotoxic. Botulinum is one of the most deadly toxins.
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Answer:
Its 5.3 litres per minute
Explanation:
___________
Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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Answer:
2.5 (B)
Explanation:
because if you multiply 5 and 2.5 it equals 12.5
