B. Plasmid hope this helps.
The correct answer would be spilled diesel fuel
<h2><u>
Full Question:</u></h2>
In the family tree below, people with the recessive trait of attached earlobes are shaded gray.
What must be true about the person labeled "A"?
A. It is a male with at least one dominant allele.
B. It is a male with two dominant alleles.
C. It is a female with at least one dominant allele.
D. It is a female with two dominant alleles.
<h2><u>Answer:</u></h2>
Its a male with atleast one dominant allele.
Option A.
<h3><u>Explanation:</u></h3>
The gene for the attached earlobe is recessive while the gene for the free earlobes is dominant. In the phylogenetic tree, we can see that both the father and mother aren't having attached earlobes. So both of them are having atleast one dominant allele which makes them have free earlobe.
In the F1 offsprings, one of the female and a male is having free earlobes. So both of them have atleast one dominant allele. The 2nd female is having an attached earlobe. So both the recessive allele have come form one parent each. So both of them are heterozygous.
Thus, the male marked as A atleast have one dominant allele. He can be a homozygous dominant, but the probability is 25%.
There are a variety of points in the transcriptional chain at which it is possible to disrupt protein synthesis in bacteria. Let’s enumerate just a few:
<span>There’s the initial point where DNA is transcribed into mRNA;<span>there’s the point where mRNA binds to the Ribosome complex;</span>there’s the point where tRNA-aminoacyl pair binds to the Ribosome according to the current codon being “read out” in the mRNA;there’s the point where the aminoacid transported by the tRNA is transferred to the growing protein chain; andthere’s the point where the protein synthesis is determined complete, and the Ribosome disengages and releases the newly-synthesized peptide chain.</span>
In each of these stages (and in some other, more subtle phases) there are possible points of disruption and there are specific disruptors; some of which are indicated in the aboveProtein synthesis inhibitor article.
Note, by the way, that the Ribosomes of Prokaryotes (bacteria) and Eukaryotes (cells with nuclei) aren’t identical, and therefore the inhibitors/disruptors that work for one type of cell may not (and usually don’t) work on the other type. That’s why we can take antibiotics targeted at bacteria with little to no fear of them interfering with our eukaryotic cells’ functions.
(This is a simplified, somewhat hand-wavy response. There is a lot more to say, mainly because biological systems are anything but simple. Nevertheless this should be enough to get you started in the general direction.)
