Answer: Generally, these factors are based on two things: hydrostatic pressure and osmotic pressure. Water will only flow across a membrane firstly if the membrane is permeable to water.
Explanation:
Creatinine is a by-product of muscular metabolism. In the natural and normal scheme of things, this substance or waste product can be eliminated from the body. A high-serum creatinine level may cause kidney damage. In relation to the above question as to how high can creatinine levels go before death, it must be noted that kidneys have strong compensatory ability and by that as long as its still 50 percent functional, creatinine level won't be that high. Which leads us to a conclusion that, the lesser the kidney function level is, the higher the creatinine level.
On the other hand for the blood urea nitrogen (BUN) is the most stable blood constituent following death as it reaches antemortem (before death) levels and even after moderate decomposition.
Therefore, as long as the kidneys are functional, regardless of other factors such as the patient's condition/ diagnosis, these blood constituents are nearly normal.
Answer:
25% or 1/4
Explanation:
The gene for colour in Heliodors is controlled by two contrasting alleles that codes for Red (R) and Yellow (Y) colours. However, these two alleles exhibit incomplete dominance, which is a phenomenon whereby a combination of both alleles gives rise to a third intermediate phenotype that is a blending of the other two parental phenotypes. In this case, both colours gives rise to a heterozygous Orange coloration (RY) in Heliodors.
However, if two orange Heliodors (RY) are crossed, four possible offsprings will be produced with the genotypes: RR, RY, RY, YY. This shows a phenotypic ratio of 1 red: 2orange: 1yellow. Hence, the probability of having a child with red coloration is 1 out of 4 possible offsprings i.e. 1/4.
Expressing this in percentage, we have 1/4 × 100 = 25%.
Answer:
No changes occur to the K1 value, its concentration remains higher than the concentration of the inhibitor and enzyme inhibitor complex
Explanation:
Uncompetitive inhibition is an example of a reversible inhibition. Reversible inhibitors bind to enzymes by weak non-covalent bonding. Thus the formation and dissociation of this association is rapid. uncompetitve inhibition lowers the Vmax and Km.
