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a) when lactose is absent, the repressor is active, RNA Polymerase cannot bind to the promoter and the structural gene are not formed.
b) when lactose is present, the lac repressor is inactive, RNA Polymerase can bind to the promotor and the structural genes are formed.
Explanation:
The lac operon in E.coli has genes required for lactose metabolism.
It is expressed only when lactose is present in the medium and glucose is absent.
According to the glucose and lactose level the operon is switched on or off.
The lac repressor or allolactose and catabolite activator protein is present in lac operon.
Glucose is the primary source of energy but bacteria loves to fuel down lactose. To use lactose as an energy source lac operon works.
a) when lactose is absent, the repressor is active, RNA Polymerase cannot bind to the promoter and the structural gene are not formed.
b) when lactose is present, the lac repressor is inactive, RNA Polymerase can bind to the promotor and the structural genes are formed.
The lac repressor is a protein that inhibits transcription of lac operon in the absence of lactose.
Answer:
The correct answer is a acyl-carnitine readily cross the mitochondrial inner membrane but the acyl CoA do not.
Explanation:
Fatty acids are activated to form fatty acyl CoA by the help of ATP and CoA SH.But the so formed Acyl CoA cannot cross or pass the inner mitochondrial membrane to enter the mitochondrial matrix to initiate beta oxidation of fatty acid.
To overcome this problem fatty acyl CoA is converted to acyl carnitine and the so formed acyl carnitine readily crosses the inner mitochondrial membrane and enters the mitochondrial matrix.
Answer:
Mutations in the DMD gene
Explanation:
Mutations in the DMD gene alter the structure or function of dystrophin or prevent any functional dystrophin from being produced. Muscle cells without enough of this protein become damaged as muscles repeatedly contract and relax with use.
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