Competitive inhibition vs allosteric inhibition
In competitive the substrate and inhibitor bind at the same active site - pretty straightforward. In allosteric regulation (speaking specifically about inhibition here), the inhibitor is binding at a site other than the active site, and changing the enzyme in some way to make it inactive.
Answer:
Explanation:
The host's immune response to the transplanted graft, which is commonly exhibited as pericapsular fibrotic overgrowth (PFO), is one of the key causes of defective encapsulated islets in (PFO).PFO creates a barrier upon this capsule surface that inhibits and impedes oxygen and nutrients from entering, resulting in islet cell deprivation, hypoxia, and/or death. This host immune response was missing under in vitro circumstances, which explains why glucose sensing and insulin release were more efficient than in vivo circumstances. Nonetheless, utilizing nanoporous encapsulation or modifying the microcapsular shape and geometry can solve these issues.
Answer:
the cell cycle
Explanation:
Image of the cell cycle. Interphase is composed of G1 phase (cell growth), followed by S phase (DNA synthesis), followed by G2 phase (cell growth). At the end of interphase comes the mitotic phase, which is made up of mitosis and cytokinesis and leads to the formation of two daughter cells.
Answer:
A a living thing that has undergone a mutation.
Explanation: