Answer:
The autonomic nervous system is the main neural regulator of circulation and blood pressure in the short term and beat by beat and exerts its function through various reflexes that regulate vasomotor tone, heart rate and cardiac output. At the renal level, the renin–angiotensin–aldosterone system is possibly the most important in the maintenance of arterial homeostasis.
Explanation:
Blood pressure is regulated by a series of interrelated autonomic systems and humoral reflexes, which continually adjust the determining elements of the system (heart rate, stroke volume, total peripheral resistance and circulating volume).The effective circulating volume is controlled by a series of reflex systems, which obtain information about the perfusion pressure (baroreceptors in the carotid bulb and aortic arch), plasma osmolarity (hypothalamus) and urinary sodium (distal tubule).The kidney has its own self-regulatory mechanisms. The reduction in renal blood flow is detected at the level of the mesangial cells of the juxtaglomerular apparatus, starting the renin-angiotensin system. The increase in angiotensin II produces on the one hand local vasoconstriction, and on the other hand stimulates the production of aldosterone by the adrenal cortex with the consequent tubular reabsorption of sodium and water.Antidiuretic hormone or vasopressin (released from the hypothalamus by stimulation of arterial baroreceptors and also by stimulation of angiotensin II) also acts at the renal level, which acts as a powerful and water-saving vasoconstrictor in the distal tubule.
3 types of energy that could be used in my house would be solar, wind, and normal electric. The ideal energy source that is not harmful to the environment is solar, as it can power a house, and doesn't use up non-renewable resources.
Brainliest, please?Thanks!
Answer:
200 approximate
Explanation:
depends how many in common
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
Answer: its to overturn such resiliency
Explanation:
