Because Muscles that are inactive have a higher incidence of venous stasis.
Venous stasis can occur when the muscles of the extremities are inactive. Venous stasis is a risk factor in Virchow's triad.
<h3>What Is Venous Thromboembolism?</h3>
Venous thromboembolism (VTE), additionally called blood clots, is a sickness that consists of deep vein thrombosis and pulmonary embolism. A deep vein thrombosis (DVT) takes place while a blood clot bureaucracy in a deep vein, typically withinside the decrease leg, thigh, or pelvis.
Your risk of developing VTE is highest after major surgery or serious injury, or when you have heart failure, cancer, or a heart attack. Swelling, redness, and pain are some of the symptoms of deep vein thrombosis. Pulmonary embolism can cause sudden chest pain and shortness of breath.
VTE sometimes occurs without any obvious signs. Medicines to help prevent more blood clots from forming or to clear up severe vein blockages are the mainstay of treatment for VTE.
To learn more about venous thromboembolism (VTE) from the given link
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It is also called the Ellipsoidal Joint
Answer:
Some dentists may choose to become dental surgeons or orthodontists. This requires more time in residency, however, more time in medical school is nto required.
Explanation:
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
Answer:
About a pound
Explanation:
7 days * 500 cal / day = 3500 cal <==== about a pound of fat cals
