Answer: D) A young tadpole has gills, a tail and no legs
The answer should be C, progesterone.
Progesterone is the hormone which can help maintain the lining of uterus and to further thicken it so that the uterus is prepared for implantation and the development of a baby. The level of progesterone is significantly rose after ovulation.
On the other hand, LH, luteinising hormone, is produced by oestrogen, another female hormone which will drop after ovulation. LH helps stimulate ovulation and prevent the development of other follicles, the origin of eggs, to form yellow body instead. Which yellow body is the one which stimulates the production of progesterone.
Last but not least, I believe D should be testosterone, and it is a male hormone so it will not be found in female normally.
Therefore, your answer should be C, progesterone.
Answer:
When lichens and misses died off, they are usually replaced by bigger plants because they have made the place suitable for the growth of larger plants. The dead parts of lichen and misses decompose and turn into small particles of soil, this allow other plant species to grow in this location.
Answer:
Ribosomes are made up of macromolecule "nucleic acids " especially RNA ( ribonucleic acid)
* More than 40 proteins and glycoproteins involved in the complement system are synthesized by the liver, macrophages, epithelial cells, they are present in the blood in plasmatic form, membrane, some have an enzymatic activity, regulator or membrane receptorThese are elements of the humoral innate immune response, they fight infections, purify immune complexes and apoptotic bodies.
<span>There are indeed three ways to activate the complement:</span>
Classical pathway: Activated by Immunoglobulins in immune complexes, aggregated Immunoglobulins, DNA, CRP, apoptotic bodies .......it involves nine fractions, starting with C1, then C4, C2, C3, to form a classical C5 convertase, then, activation of C5, C6, C7, C8, C9.
Alternative pathway: activated by polysaccharides (bacterial endotoxin), vascular wall poor in sialic acid, aggregated IgE ...C3b like is the first component in the alternate channel cascade, it will create an amplification loop, and form an alternative C5 convertase.
Lecithin pathway: Activated by mannose, fucose (carbohydrate of microorganisms)The first component is the complex MBL / MASP1 / MASP2: "mannose-binding protein": works according to the same principle as the complex C1 of the classical way (MASP2 cleaves the C4 and the rest of the cascade is equivalent to that of the classical way).
the three ways have the same outcome: A C5 convertase (formed by one of the pathways) cleaves C5 into C5a and C5b: C5b is deposited far from other fractions on the antigenic surface. The fixation of C5b in the cell is followed by that of C6, C7, C8, and C9 (9 molecules of C9): formation of the membrane attack complex (MAC) ==> Death of the cell by osmotic shock
