It is true but commonly called Richter meter
Answer:
He should get his mitochondrial haplotype sequenced, given the genetic variation in mitochondrial sequences compared to Y chromosomes.
Explanation:
- Mitochondrial DNA haplotypes are specific regions of mitochondrial DNA that cluster with other mitochondrial sequences to show the phylogenetic origins of maternal lineages.
- Mitochondrial DNA haplotypes are associated with a range of phenotypes and disease.
- Mitochondrial DNA contains 37 genes, all of which are essential for normal mitochondrial function.
- Thirteen of these genes provide instructions for making enzymes involved in oxidative phosphorylation.
When was BSE first discovered?
BSE first appeared in British cattle in the mid-1980s. There have been numerous cases detected in other countries. A complete list of countries is available from both the U.S. Food and Drug Administration (FDA) and the U.S. Department of Agriculture (USDA).
Answer:
the first one is diploid
meoisis produces haploid gametes that have half the number of chromosomes (23).
not sure about the rest
Explanation:
Answer:
C) Through genomic imprinting, methylation regulates expression of the paternal copy of the gene in the brain.
Explanation:
The pattern of gene expression wherein either paternal or maternal gene is expressed in specific cells while the other one is prevented from expression is known as genomic imprinting.
In the given example, the maternal copy of the gene on chromosome 15 is expressed in brain cells while its paternal copy is not expressed in these cells. Hence, the pattern of expression of this gene is regulated through genome imprinting. One of the mechanism is methylation of cytidine residues of CpG islands of the DNA that are more frequently present within promoters of the genes.
When the cytidine residues of these sequences are methylated into 5-methylcytidine, the transcription factors do not bind to these promoters preventing the expression of these genes.
Hence, methylation of cytidine residue in CpG islands of the promoters of the gene present on chromosome 15 could have silenced its expression in brain cells.
