Answer:
This results in a third phenotype in which the expressed physical trait is a combination of the phenotypes of both alleles.
Explanation:
an example is like if a red and white flower create another flower the flower will be pink because both alleles are dominant and will be phenotypically expressed.
Areoles are the tissue that make up most of the stem of cacti. These are specialized structure that give birth to flowers and are often tubular and multipetaled. This structure enables the plant to survive in the harsh conditions of the desert and help them absorb water from the soil efficiently.
Answer: It is possible for two O bloody type to have a child of Blood type B which can be caused by mutation.
Mutation is the changes in genetic content or DNA . Although it is normal for two O blood type parents to have O type child but it is technically possible for O blood type parents to have child of A or B blood type which is due to changes in DNA. Changes in DNA can be caused when sunlight hit our bodies, conversion of food to energy , when our body absorb some chemicals and when our cells try to copy another DNA. The differences between the blood types A, B, and O is very small. This makes it possible for a mother of O blood type produce B egg or a father of O blood type produce B sperm which is caused by mutation.
For a parents with blood type O to have a child with B type blood, the parents blood type O would have to look like B type .
Explanation:
Mutation is the change in genetic material or DNA. It can lead to change in blood type of humans with different parents blood types. This mutation can be caused by chemicals we breathe in, exposure to sunlight and so on.
Answer:
-Histamine binds extracellularly to the H1 receptor.
-When histamine binds to the H1 receptor. the receptor undergoes a conformation change and binds the inactive G protein.
-Once the G protein is active, it binds to the enzyme phospholipase C, activating it.
-Histamine is likely hydrophilic.
When histamine encounters a target cell, it binds extracellularly to the H1 receptor, causing a change in the shape of the receptor. This change in shape allows the G protein to bind to the H1 receptor, causing a GTP molecule to displace a GDP molecule and activating the G protein. The active G protein dissociates from the H1 receptor and binds to the enzyme phospholipase C, activating it. The active phospholipase C triggers a cellular response. The G protein then functions as a GTPase and hydrolyzes the GTP to GDP. The G protein dissociates from the enzyme and is inactive again and ready for reuse.
Explanation:
