Answer:
Gal4p is a transcription factor that targets specific GAL genes, while Gal11p is a core component of the Mediator complex that plays regulatory roles in the expression of many different genes (class II genes) in eukaryotic organisms
Explanation:
The Gal4 transcription factor is one of the most widely studied eukaryotic transcriptional activators capable of activating the expression of galactose-induced genes. Gal4 is well-known to target specific enzymes involved in galactose metabolism: GAL1, GAL7, and GAL10. On the other hand, Gal11p is a key subunit of the evolutionary conserved Mediator complex that binds to the RNA polymerase II (RNAP II) in order to regulate the transcription of class II genes (i.e., genes transcribed by RNAP II).
Answer:
The correct answer is "I would suggest her to change the Gram positive control from Bacillus subtilis to other bacteria".
Explanation:
Bacillus subtilis, and the species from the genre Bacillus, are known for losing the crystal violet coloration during Gram testing. It is likely that B. subtilis might look pink under the procedure for this reason. I would suggest to change her Gram positive control from B. subtilis to other bacteria. For instance, she could try with Staphylococcus aureus, a widely used bacteria as Gram positive control.
To avoid this u need to drink LOTS of water
An organic compound consists of carbon...
The origin of congenital adrenal hyperplasia occurs when circulating cortisol levels are low.
Congenital adrenal hyperplasia (CAH) is an autosomal recessive disease which results from mutations of genes for enzymes involved in the biochemical steps of production of mineralocorticoids, glucocorticoids or sex steroids from cholesterol by the adrenal glands (steroidogenesis). <span>Low cortisol production results in rising levels of ACTH (adrenocorticotropic hormone) because cortisol usually inhibits ACTH production. This increased ACTH production induces overgrowth (hyperplasia) and overactivity of the steroid-producing cells of the adrenal cortex.</span>
