Mendel was able to attribute the variation observed in the offspring of his experiment to the controlled fertilization process.
Mendel was able to control pollination and, thus, the fertilization process in the pea plants used for his experiment.
Mendel was able to effectively predict the outcome with self-pollination or cross-pollination with different results coming from each. Thus, he logically concluded that the variation observed in the offspring of his crosses is due to the fact that he controlled the fertilization process.
More on Mendel's experiments can be found here: brainly.com/question/3186121?referrer=searchResults
Answer:
Both the volume of water and the rate of erosion will increase.
Explanation:
Answer:
For the Numbers on the leg Photo
1. Rectus Femoris
2. Vastus lateralis
3. Tibialis Anterior
4. ADDuctor longus
5. Gracilis
6. Satorius
7. Vastus Medialis
8. Gluteus Medius
9. Gluteus Maximus
10. Semitendinosis
11. Semimembranosus
12. Biceps Femoris
13. Gastrocnemius
Explanation: is correct
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If you look at a zipper, you'll see that it doesn't have a wheel or an axle. A zipper links the <span>protruding teeth that the zip contains. Your answer would be B!
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Answer:
A. His mitochondria lack the transport protein that moves pyruvate across the outer mitochondrial membrane.
Explanation:
Pyruvate is from the breakdown of carbohydrates such as glucose through glycolysis. Glucose enters the cytosol through specific transporters (the GLUT family) and is processed by one of several pathways depending on cellular requirements. Glycolysis occurs in the cytosol and produces a limited amount of ATP, but the end product is two 3-carbon molecules of pyruvate, which maybe diverted again into many pathways depending on the requirements of the cell. In aerobic conditions, pyruvate is primarily transported into the mitochondrial matrix and converted to acetyl-coenzyme A (acetyl-CoA) and carbon dioxide by the pyruvate dehydrogenase complex (PDC).
Initially it was proposed that pyruvate was able to cross the membrane in its undissociated (acid) form but evaluation of its biochemical properties show that it is largely in its ionic form within the cell and should therefore require a transporter.
Transport of pyruvate across the outer mitochondrial membrane appears to be easily accomplished via large non-selective channels such as voltage-dependent anion channels/porin, which enable passive diffusion. Indeed, deficiencies in these channels have been suggested to block pyruvate metabolism
