Answer:
CPR.
Explanation:
Because when the client/patient is been rushed to the ED with pulmonary edema they won't breathe if anyone doesn't save them when they need oxygen at the most urgently needingless times.
If Mr. Jones would like a refill on his flomax® prescription because his prescription expired. You should first ask the doctor to check his body again to see if he needs more medication or not.
<h3>What is a physician?</h3>
A physician is a person who has a master's degree in medicine, and he can treat the basic problem of the whole body. He can prescribe you medicines and treatment for basic health issues.
Thus, If Mr. Jones requires a new prescription for flomax® because his old one has run out. To determine whether he requires extra medication, you should first request that the doctor perform another physical examination of him.
To learn more about physician, refer to the link:
brainly.com/question/17958491
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Answer:
The mitral valve has fewer flaps than the right atrioventricular valve:
<em>a) True </em>
Explanation:
The mitral valve is a bicuspid valve, which means it has two flaps; whereas, the right atrioventricular valve is a tricuspid valve and so has three flaps.
These two heart valves link the atrium with the ventricles. The mitral valve is located on the left side of the heart, while the tricuspid valve is on the right side.
Answer:
b) blastic red blood cell (RBC).
Explanation:
In excess of 340 blood group antigens have now been described that vary between individuals. Thus, any unit of blood that is nonautologous represents a significant dose of alloantigen. Most blood group antigens are proteins, which differ by a single amino acid between donors and recipients. Approximately 1 out of every 70 individuals are transfused each year (in the United States alone), which leads to antibody responses to red blood cell <u>(RBC) alloantigens</u> in some transfusion recipients. When alloantibodies are formed, in many cases, RBCs expressing the antigen in question can no longer be safely transfused. However, despite chronic transfusion, only 3% to 10% of recipients (in general) mount an alloantibody response. In some disease states, rates of alloimmunization are much higher (eg, sickle cell disease). For patients who become alloimmunized to multiple antigens, ongoing transfusion therapy becomes increasingly difficult or, in some cases, impossible. While alloantibodies are the ultimate immune effector of humoral alloimmunization, the cellular underpinnings of the immune system that lead to ultimate alloantibody production are complex, including antigen consumption, antigen processing, antigen presentation, T-cell biology.
